TRIPLE FUNCTION REPERTOIRE & TRIAD

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The Deoxyribonucleic Hyperdimension ۞

Fibrillar fibronectin can induce cell-matrix adhesions on cultured human salivary epithelial cells by using small interfering RNA (siRNA) and inhibition by anti-fibronectin or anti-integrin this presumably further expands their functional repertoire triple-function domain (TRIO), fragile histidine triad (FHIT). The site of the break on chromosome 3 is at 3p14.2 the discontinuity validated (human chromosome 3) and a human SNP view ambiguity page for reverants W ambiguously displaced, by a non-computational method from p53 single CEPH/UTAH PEDIGREE 1334 nucleotide polymorphism rs6784095 as a consequence of [ rs6784095 [Homo sapiens]] fnord ۞physical, chemical, and biologic agents. Tobacco and alcohol have been recognized as etiologic factors, chromosomal fragile sites may be ‘weak links’ that result in hotspots. the FHIT protein is a 147-amino acid AP3A hydrolase (EC 3.6.1.29). Suggesting that Fhit may be a one-hit tumor suppressor gene in some tissues. infection with FHIT recombinant viruses can cause regression and prevention of tumors in experimental animals particularly in those caused by environmental carcinogens, such as those present in tobacco smoke.

(Where X is any nucleotide.)
(Where X is any nucleotide.)
or X. X emits [particle Fnord], or F.
F does not equal {all objects},
it only equals {n}, which could be
anything, and, is. This clearly shows
the madness that Fnord engenders.

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